Homotoxicology

What is Homotoxicology?

Homotoxicology studies the influence of toxic substances on humans. Dr Hans-Heinrich Reckeweg, the German medical doctor and homoeopath who developed Homotoxicology and referred to these substances as homotoxins.

In Homotoxicology all symptoms and disease's are viewed as a result of the appropriate, biological response, or resistance to toxic substances (homo-toxins).

When the body is unable to excrete toxins or acids, the healing phase of inflammation becomes active to try to rid the body, or the area of the toxins. If the increase in heat, or more commonly called inflammation cannot clear the toxin, the body will deposit the toxins somewhere in the body where they will not cause so much of an issue for homeostatic mechanisms. In other words, these toxins will be "walled off and isolated" from the rest of the body's communication systems. 

If the acid or toxin that was trying to be excreted could not be expelled, it backs up more and is deposited deeper and deeper into the system. In deeper, more vital organs such as the arteries, the heart, or lung. In short the toxin or homotoxin will get deposited at the weakest organ of the body, the locus minoris resistentiae. When the homotoxin cannot be excreted the body deposits it.

This is the basis of Homotoxicology that Dr Hans-Heinrich Reckeweg theorized in 1955 as a unifying approach of synthesising medicine and Homeopathy. 

The guiding diagnostic and prescriptive tool used in homotoxicology is known as the Six-Phase Table. The table allows the allocation of symptoms to the defined phases of the body's response and thereby assists in the facilitation of the appropriate preparations.

Diseases in Homotoxicology are expressions of the vital life force of a living organism against toxins, and its efforts to counteract and expel them.
— John Bumpus

 

In Nutritional Balancing, when the body is exhausted and depleted of energy, to the point that it can no longer expel homotoxins, for whatever reason, the organism tries through increased pathological means to make up for the damage already sustained.

This process goes in six distinct phases:

1. Excretion phase - or the expulsion of toxins through body orifices.

e.g. In this phase the body's defensive system is intact and can excrete homo-toxins in various ways such as through diarrhoea or a runny nose, fever, inflammation, vomiting; and mobilisation of white blood cells to consume the toxins.

2. Reaction phase - Where toxins are removed by the body responding to homotoxins, If excretion is not sufficient, the body has an inflammatory response (such as a fever) in an attempt to neutralise toxins. 

e.g. fever, inflammation and mobilisation of white blood cells to consume the toxins.

3. Deposition phase - storage followed by deactivation of the toxins in connective and fat tissue and the vascular system.


If homo-toxins are not sufficiently excreted and continue to flow into the body, the toxic products are stored in the extracellular space. This phase often occurs without symptoms or indicators.

The above phases are naturally "reversible". The following phases become more and more difficult to deal with as in these processes damage occurs to the organs themselves.


4. Impregnation phase - severe disease or imbalance occurs in a "locus minoris resistentiae", the body's weakest organ; Abundant toxins within the cells destroy large cellular groups within an organ, resulting in organ degeneration. 

Toxins have invaded the cell, and the toxins themselves become part of the connective tissue and matrix. Increasingly severe symptoms are typical of this stage and indicate damage to organ cells.

5. Degeneration phase - Organ is increasingly and "irreversibly damaged".

Alteration of the cellular enzymes and in the organic structure; Abundant toxins within the cells destroy large cellular groups within an organ, resulting in organ degeneration.

6. Differentiation phase - Genes in the cells are damaged. Illnesses, in this phase, are characterised by the creation of undifferentiated, non-specialized cell forms.